[Ventricular dysfunction in obstructive sleep apnea-hypopnea syndrome: in search of clinical significance].
نویسندگان
چکیده
Obstructive sleep apnea-hypopnea syndrome (OSAHS), characterized by repetitive episodes of complete or partial interruption of the airflow during sleep, constitutes a growing healthcare problem. Prevalence in the general population is high and resulting morbidity and mortality, principally attributed to traffic accidents and the development of cardiovascular complications, quite considerable.1 Although most epidemiologic evidence corresponds to the association of OSAHS with high blood pressure, cerebrovascular disease, and heart failure,2,3 it has also been associated with cardiovascular disturbances like ischemic heart disease, cardiac arrhythmias, or pulmonary hypertension.3-6 Moreover, the identification and definition of possible pathogens, and the positive effects of treatment on the cardiovascular system have strengthened the causal relationship. The relationship between OSAHS and heart failure is especially important due to the high prevalence of both entities and the clinical repercussions. However, we should remember that patients with OSAHS usually present other cardiovascular risk factors such as age, obesity, high blood pressure, diabetes mellitus, or ischemic heart disease, which may constitute confounding factors. Nonetheless, different ways in which OSAHS can cause systolic and diastolic left ventricular dysfunction have been recognized.2 Intermittent hypoxemia, sympathetic activation and increased ventricular preload and afterload Ventricular Dysfunction in Obstructive Sleep Apnea-Hypopnea Syndrome: In Search of Clinical Significance Miguel A. Ariasa and Francisco García-Ríob
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عنوان ژورنال:
- Revista espanola de cardiologia
دوره 60 6 شماره
صفحات -
تاریخ انتشار 2007